By Robert S.; Thimann, Kenneth V.; Marrian, G. F. Harris
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Extra info for Vitamins and Hormones, Vol. 13
11. Chemical Relations of Polyunsaturated Fatty Acids.. 80 2. Methods of Preparation of Polyunsaturated Acids . . . . . . . . 32 c. The Urea-Adduct Method.. . . . . . . . . . . . . . . . 32 d. Preparation from Bromo-Derivatives. . . . . . . . . . . . . . 33 e. Chromatographic and Related Methods.. . 3. Chemical and Physical Methods for the Analy Acids. . . . . . d. “Isolation” Method of Simmons and Quackenbush..
BIO-ASSAYOF ESSENTIAL FATTY ACIDS 1. , 1950). From then on, the body weight may remain fairly constant over an extended period of time, or it may gradually decline, and the rats may succumb. If EFA are fed to the depleted rats on a basal fat-free diet after a constant body weight has been established, an increase in body weight promptly occurs. Within certain limits, the increase is proportional to the amount and potency of the active compound employed. Greenberg and associates (1950) reported that, when the log dose of linoleate fed was plotted against the gain-in-weight of male rats for doses of linoleate of 5, 10, 20, or 50 mg.
1942) could be utilized in these studies. The experiments described above suggest that the decreased level of circulating antibodies in the vitamin deficiencies cannot be attributed to a faulty release mechanism or to an excessive destruction of antibody. It seems more likely that there has been a disturbance in the processes of antibody synthesis. For the present the question of the direct participation of the vitamins in antibody production must remain sub judice. V. SUMMARY The data presented in this review have demonstrated clearly that an antibody response can be markedly inhibited in various vitamindeficiency states.
Vitamins and Hormones, Vol. 13 by Robert S.; Thimann, Kenneth V.; Marrian, G. F. Harris