Bratton's Family Medicine Board Review - download pdf or read online

By Robert L. Bratton

ISBN-10: 0781772877

ISBN-13: 9780781772877

Thoroughly up-to-date for its 3rd variation, this e-book is a complete evaluation for the yank Board of family members medication certification and recertification assessments. This version comprises over 1,800 board-format questions, together with over 1,000 multiple-choice questions from the foremost topic components of kin perform and over seven-hundred questions drawn from 60 scientific challenge units. The booklet encompasses a pictorial atlas of medical images, radiographs, and lab smears, with questions concerning those photos. specific solutions and motives stick to the questions.

This booklet comprises 20 AMA PRA class 1 Credit(s)™ backed via Lippincott Williams & Wilkins.

Four perform assessments can be found on a significant other Website.

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Otras pruebas de apoyo ayudan a aclarar la variante específica de acidosis con desequilibrio aniónico (véase más adelante). ■ ACIDOSIS METABÓLICA La concentración baja de bicarbonato en la acidosis metabólica se debe a la adición de ácidos (orgánicos o inorgánicos) o a la pérdida de HCO3–. Las causas de la acidosis metabólica suelen clasificarse con base en la presencia o ausencia de aumento de el desequilibrio aniónico (cuadro 2-6). La acidosis con aumento de desequilibrio aniónico (>12 mmol/L) se debe al aporte de ácido (distinto al HCl) y aniones no medidos.

Diabetes, nefropatía diabética 3. Fármacos: fármacos antiinflamatorios no esteroideos (NSAID), inhibidores de la ciclooxigenasa 2 (COX-2), bloqueadores beta, ciclosporina, tacrolimús 4. Nefropatía crónica, edad avanzada 5. Seudohipoaldosteronismo de tipo II: defectos en las cinasas WNK1 o WNK4 Electrólitos y equilibrio acidobásico CAPÍTULO 2 15 CUADRO 2-4 Causas principales de hiperpotasemia (Continuación) D. Resistencia renal a mineralocorticoide 1. Enfermedades tubulointersticiales: SLE, amiloidosis, anemia drepanocítica, uropatía obstructiva, síndrome posterior a necrosis tubular aguda 2.

Véase el texto para detalles. BP, presión arterial; DKA, cetoacidosis diabética; FHPP, parálisis periódica hipopotasémica familiar; FH-I, hiperaldosteronismo familiar de tipo I; GI, gastrointestinal; HTN, hipertensión; PA, aldosteronismo primario; RAS, estenosis de arteria renal; RST, tumor secretor de renina; RTA, acidosis tubular renal; SAME, síndrome de exceso aparente de mineralocorticoides; TTKG, gradiente transtubular de potasio. [Tomada con autorización a partir de Mount DB, Zandi-Nejad K.

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Bratton's Family Medicine Board Review by Robert L. Bratton


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