New PDF release: Biology and Pathology of Astrocyte-Neuron Interactions

By Leif Hertz (auth.), Sergey Fedoroff, Bernhard H. J. Juurlink, Ronald Doucette (eds.)

ISBN-10: 147579486X

ISBN-13: 9781475794861

ISBN-10: 1475794886

ISBN-13: 9781475794885

This quantity is made from papers provided on the moment foreign Altschul Symposium: Biology and Pathology of Astrocyte-Neuron Interactions. The symposium used to be held in Saskatoon, Canada on the collage of Saskatchewn in may well, 1992 in reminiscence of Rudolf Altschul, a graduate of the collage of Prague and a pioneer within the fields of the biology of the vascular and apprehensive platforms. Dr. Altschul used to be Professor and Head of the dep. of Anatomy on the college of Saskatchewan from 1955 to 1963. The Altschul Symposia have been made attainable by means of an endowment left by way of Anni Altschul and via different contributions. The symposia are held biennially. one of many maximum demanding situations for trendy scientists is to discover the mechanisms of mind functionality. even if mobile anatomy of the frightened method has already been good defined and certainly used to be delineated by way of the start of the century, experimental research of the functionality of the mind is comparatively fresh. The framework of the mind is made from stellate cells, the astrocytes, that are interconnected through their methods, therefore featuring a meshwork by which the neurons ship their axons, observed through oligodendrocytes. Microglia are disbursed during the brain.

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13:21-47. , 1986, Electrophysiological properties of astrocytes, in: "Astrocytes", S. Federoff and A. , Academic Press. London. , 1984, The rat optic nerve following enucleation: a pure population of mammalian glia. Abstr. Soc. Neurosci. 10:949. , 1990, Neuroaminidase treatment modifies the function of electroplax sodium channels in planar lipid hi-layers, Neuron 5: 675-684. , 1987, Cytotopographical specialization of enzymatically isolated rabbit retinal Muller cells: K+ conductance of the cell membrane, Glia, 1: 191-197.

Following early demonstrations of Na+ channels in cultured astrocytes, Ritchie and his colleagues (Gray and Ritchie 1985; Bevan et al. 1985) suggested the possibility that astrocytes might function as subsidiary sites for the synthesis of sodium channels which are transferred to the adjacent axon membrane. Cell-to-cell transfer of ion channels has not been demonstrated to date. Thus the idea, that astrocytes synthesize sodium channels destined for transport to the axon, remains speculative. Although astrocyte-to-axon transfer of macromolecules has not yet been demonstrated in mammals, recent experiments have demonstrated that mammalian astrocytes can express Na+ channels with biophysical properties similar to Na+ channels expressed by neurons (Barres et al.

Proton Shifts in WM are Much Smaller than Those in GM Several factors may account for the smaller size of the anoxic acid shift in WM in comparison with GM. First, synapses and SD-like events in GM may have a significant impact on membrane permeability, as was outlined previously. , 1990), and the ion channels that open in spreading depression probably facilitate transmembrane proton fluxes (Kraig and Nicholson, 1978), the absence of these processes in WM could account for its smaller anoxic L1pH0 , just as they help to explain its diminished [M{+]o.

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Biology and Pathology of Astrocyte-Neuron Interactions by Leif Hertz (auth.), Sergey Fedoroff, Bernhard H. J. Juurlink, Ronald Doucette (eds.)


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